Where to Buy APINACA (AKB-48) Online
What is APINACA (AKB-48) ?
5C-AKB48 is an indazole based cannabinoid that is the 5-chloro analogue to 5F-AKB48 and is expected to produce similar results. Buy 5C-AKB48 Online.
Therefore 5C-AKB48 is a more potent form of AKB-48, which acts upon the cannabinoid receptors and also is suitable for a comparable study into the effects agonists upon the CB2 receptors.
|Synonyms:||5-CHLORO APINACA, 5C-APINACA|
|Molecular weight:||399.96 G/MOL|
Therefore its abuse has been associated with multiple serious injuries deaths and more dangerous side effects and toxicity in general. Buy 5C-AKB48 Online.
Where to buy 5F-AKB48 online?
Also, we have the purest form of 5F-AKB48. Although this substance has not been formally studied, from analysis of the structure.
However is presumed that 5F-AKB48 has a similar binding profile to that of other cannabinoids and matches many of the in vivo properties of Δ9-THC. However, the role of these interactions and how they result in the cannabinoid high continues to remain elusive.
So, 5F-AKB48 (also known as 5F-APINACA and AKB-48F) is a synthetic cannabinoid substance of the indazolecarboxamide class.
Nevertheless it acts as a potent agonist for the CB1 and CB2 cannabinoid receptors. It was first identified in South Korea and is available for sale as a grey area research chemical through online vendors.
However 5C-AKB48 is a more potent form of AKB-48, which acts upon the cannabinoid receptors, and is a key component of many synthetic cannabis blends.
Also 5C-AKB48Â is suitable for comparable study into the effects of banned agonists upon the CB2 receptors.
Therefore BAC offers 5C-AKB48 for the purposes of research only and also it not for recreational use or human consumption.
Above all it produces subjective effects somewhat similar to that of cannabis with a short duration and also an emphasis on intense physical sensations.
Also its metabolism has been described in the scientific literature.
Above all 5C-AKB48 is suitable for comparable study into the effects of banned agonists upon the CB2 receptors.